This substance is decreased in neurodegeneration and Alzheimer's disease.
There is especially 1 substance which is very important for the brain and the nervous system, and that is glutathione, a very effective antioxidant as well as an immune system regulator. Glutathione is a substance the body is producing, and it is involved in many functions throughout the body. The body is however producing less as we get older, and that's when we need more.
In the brain glutathione also regulates cell signaling, protein function, gene expression, and cell differentiation/proliferation.
Decreased levels of glutathione and increased oxidative stress are common in major neurodegenerative diseases (Ayoama K, Nakaki T, 2013).
In the following study proton magnetic resonance spectroscopy was used to measure brain glutathione levels in healthy controls, in patients with mild cognitive decline and in patients with Alzheimer's disease (Mandal PK, et.al., 2015). The researchers concluded that the present study provided compelling evidence that glutathione levels in specific brain regions is a clinically relevant biomarker for mild cognitive decline and Alzheimer's disease.
It is very important to do what we can early on to help prevent memory problems and Alzheimer's disease, as these neurological problems are getting more and more common.
Changes in the brain start to occur many years before any symptoms are noticed. Some years ago, glutathione could only be supplied by IV because the only oral form available, reduced glutathione is not very bioavailable, and does not produce results.
That however changed when S-Acetyl Glutathione, a patented form of glutathione, became available. S-Acetyl Glutathione gets transferred into the cells where it is needed.
Aoyama K, Nakaki T. Impaired glutathione synthesis in neurodegeneration. Int J Mol Sci. 2013 Oct 18;14(10):21021-44.
Mandal PK, Saharan S, Tripathi M, Murari G. Brain glutathione levels--a novel biomarker for mild cognitive impairment and Alzheimer's disease. Biol Psychiatry. 2015 Nov 15;78(10):702-10.
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